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2003 1
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Page 1
Good or not good: Role of miR-18a in cancer biology.
Kolenda T, Guglas K, Kopczyńska M, Sobocińska J, Teresiak A, Bliźniak R, Lamperska K. Kolenda T, et al. Rep Pract Oncol Radiother. 2020 Sep-Oct;25(5):808-819. doi: 10.1016/j.rpor.2020.07.006. Epub 2020 Aug 12. Rep Pract Oncol Radiother. 2020. PMID: 32884453 Free PMC article. Review.
AP-1-mediated chromatin looping regulates ZEB2 transcription: new insights into TNFalpha-induced epithelial-mesenchymal transition in triple-negative breast cancer.
Qiao Y, Shiue CN, Zhu J, Zhuang T, Jonsson P, Wright AP, Zhao C, Dahlman-Wright K. Qiao Y, et al. Oncotarget. 2015 Apr 10;6(10):7804-14. doi: 10.18632/oncotarget.3158. Oncotarget. 2015. PMID: 25762639 Free PMC article.
We further investigated in detail AP-1 regulation of ZEB2 expression. We show that two ZEB2 transcripts derived from distinct promoters are both expressed in breast cancer cell lines and breast tumor samples. Using the chromosome conformation capture assay, we demon …
We further investigated in detail AP-1 regulation of ZEB2 expression. We show that two ZEB2 transcripts derived from distinct …
Atypical ZFHX1B mutation associated with a mild Mowat-Wilson syndrome phenotype.
Zweier C, Horn D, Kraus C, Rauch A. Zweier C, et al. Am J Med Genet A. 2006 Apr 15;140(8):869-72. doi: 10.1002/ajmg.a.31196. Am J Med Genet A. 2006. PMID: 16532472 Review.
Mowat-Wilson syndrome is a recently delineated severe mental retardation, multiple congenital anomalies syndrome caused by dominant nonsense or frameshift mutations, deletions or translocations of the zinc finger homeobox 1B gene (ZFHX1B). We report on …
Mowat-Wilson syndrome is a recently delineated severe mental retardation, multiple congenital anomalies syndrome caused by dominant nonsense …
Genitourinary anomalies in Mowat-Wilson syndrome with deletion/mutation in the zinc finger homeo box 1B gene (ZFHX1B). Report of three Italian cases with hypospadias and review.
Garavelli L, Cerruti-Mainardi P, Virdis R, Pedori S, Pastore G, Godi M, Provera S, Rauch A, Zweier C, Zollino M, Banchini G, Longo N, Mowat D, Neri G, Bernasconi S. Garavelli L, et al. Horm Res. 2005;63(4):187-92. doi: 10.1159/000085894. Epub 2005 May 20. Horm Res. 2005. PMID: 15908750
Mowat-Wilson syndrome (MWS) is a multiple congenital anomaly syndrome characterized by a distinct facial phenotype, Hirschsprung disease, microcephaly and mental retardation. It is caused by mutations in the zinc finger homeo box 1B gene, ZFHX1B (SIP1). To da …
Mowat-Wilson syndrome (MWS) is a multiple congenital anomaly syndrome characterized by a distinct facial phenotype, Hirschsprung disease, mi …
Molecular screening of the ZFHX1B gene in prenatally diagnosed isolated agenesis of the corpus callosum.
Espinosa-Parrilla Y, Encha-Razavi F, Attié-Bitach T, Martinovic J, Morichon-Delvallez N, Munnich A, Vekemans M, Lyonnet S, Amiel J. Espinosa-Parrilla Y, et al. Prenat Diagn. 2004 Apr;24(4):298-301. doi: 10.1002/pd.865. Prenat Diagn. 2004. PMID: 15065106
Among this broad spectrum of malformations recently associated with haploinsufficiency of the zinc finger homeobox 1B gene (ZFHX1B), ACC can therefore be the only feature to be detected prenatally. ...
Among this broad spectrum of malformations recently associated with haploinsufficiency of the zinc finger homeobox 1 …
Frameshift mutation of the zinc finger homeo box 1 B gene in syndromic corpus callosum agenesis (Mowat-Wilson syndrome).
Sztriha L, Espinosa-Parrilla Y, Gururaj A, Amiel J, Lyonnet S, Gerami S, Johansen JG. Sztriha L, et al. Neuropediatrics. 2003 Dec;34(6):322-5. doi: 10.1055/s-2003-44671. Neuropediatrics. 2003. PMID: 14681759
We report a girl who had Hirschsprung disease in association with distinct facial appearance, microcephaly, agenesis of the corpus callosum and mental retardation (Mowat-Wilson syndrome). Mutation analysis of the zinc finger homeo box 1 B (ZFHX1 B) gene revealed a d …
We report a girl who had Hirschsprung disease in association with distinct facial appearance, microcephaly, agenesis of the corpus callosum …
Variations in aganglionic segment length of the enteric neural plexus in Mowat-Wilson syndrome.
Ishihara N, Shimada A, Kato J, Niimi N, Tanaka S, Miura K, Suzuki T, Wakamatsu N, Nagaya M. Ishihara N, et al. J Pediatr Surg. 2005 Sep;40(9):1411-9. doi: 10.1016/j.jpedsurg.2005.05.040. J Pediatr Surg. 2005. PMID: 16150342
BACKGROUND/PURPOSE: Patients with zinc finger homeo box 1B (ZFHX1B) mutations or deletions develop multiple congenital anomalies including Hirschsprung disease, known as Mowat-Wilson syndrome (MWS). ...
BACKGROUND/PURPOSE: Patients with zinc finger homeo box 1B (ZFHX1B) mutations or deletions develop multiple congenital …
Nonsense mutations of the ZFHX1B gene in two Japanese girls with Mowat-Wilson syndrome.
Sasongko TH, Sadewa AH, Gunadi, Lee MJ, Koterazawa K, Nishio H. Sasongko TH, et al. Kobe J Med Sci. 2007;53(4):157-62. Kobe J Med Sci. 2007. PMID: 17932455 Free article.
Mowat-Wilson syndrome (MWS) is a multiple congenital anomaly-mental retardation complex caused by mutations in the Zinc Finger Homeobox 1 B gene (ZFHX1B). MWS has been reported in association with Hirschsprung disease (HSCR). ...
Mowat-Wilson syndrome (MWS) is a multiple congenital anomaly-mental retardation complex caused by mutations in the Zinc Finger …